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In experiments of other receptor tyrosine kinases implicated during the oncogenesis of GIST, nilotinib realized strong and selective inhibition of PDGFRα and PDGFRβ. As is the situation with imatinib, nilotinib potently inhibited the autophosphorylation of A31 cells reworked by PDGFRAnilotinib will enhance the amount or influence of disopyramide